How a fat cell dies.

There are two ways that cells within the body die; necrosis and apoptosis.

Necrosis happens when the cells or the living tissues are damaged by an external influence causing an unprogrammed cell death.

When the cells die from necrosis, the cells undergo a trauma that initiates a chain reaction within the body causing a release in enzymes.  The harmful chemicals found in these enzymes can lead to further complications (such as inflammation) and damage to surrounding cells.

Apoptosis is a more clinical and natural method of programmed cell death.  This is less traumatic to the body than cell death from necrosis, Apoptosis is a more controlled and predictable cell death.

When a cell is compelled to die, which is what we trigger when performing these treatments, proteins called caspases go into action. These caspases spur on the production of enzymes known as DNases, which break down the cellular components needed for survival. In the case of  a fat cell it is through the destruction of the DNA in found in the nucleus of the cell. Once the cells have been broken down and shrunken it sends a distress signal, which is answered by the macrophages (the bodies waste disposal system). The Macrophages remove these shrunken cells by sending them to the lymphatic system for them to be removed by the body, so there is no damage caused to any of the fat cells surrounding tissue.

Cells that die from apoptosis die in response to signals within the body as opposed to cells that die from trauma (necrosis). Treatments allows us to target the fat cells and trigger apoptosis. This benefits the body by causing the destruction of these targeted cells causing cell death without inflammation of the surrounding tissue